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Estrogen receptor α (ERα) deficiency in macrophages results in increased stimulation of CD4+ T cells while 17β-estradiol acts through ERα to increase IL-4 and GATA-3 expression in CD4+ T cells independent of antigen presentation
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The selective estrogen receptor modulators, tamoxifen and raloxifene, impair dendritic cell differentiation and activation
G Nalbandian et al.
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Estrogen preferentially promotes the differentiation of CD11c+ CD11bintermediate dendritic cells from bone marrow precursors
V Paharkova-Vatchkova et al.
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T lymphocytes do not directly mediate the protective effect of estrogen on experimental autoimmune encephalomyelitis
MJ Polanczyk et al.
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Estrogen receptor α mediates estrogen's immune protection in autoimmune disease
HB Liu et al.
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The protective effect of 17b-estradiol on experimental autoimmune encephalomyelitis is mediated through estrogen receptor-a
M Polanczyk et al.
AMERICAN JOURNAL OF PATHOLOGY (2003)
Late pregnancy suppresses relapses in experimental autoimmune encephalomyelitis: Evidence for a suppressive pregancy-related serum factor
A Langer-Gould et al.
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Estrogen treatment down-regulates TNF-α production and reduces the severity of experimental autoimmune encephalomyelitis in cytokine knockout mice
A Ito et al.
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Low-dose estrogen therapy ameliorates experimental autoimmune encephalomyelitis in two different inbred mouse strains
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