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注意:仅列出部分参考文献,下载原文获取全部文献信息。Human ApoA-I transfer attenuates transplant arteriosclerosis via enhanced incorporation of bone marrow-derived endothelial progenitor cells
Yingmei Feng et al.
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Adenoviral low density lipoprotein receptor attenuates progression of atherosclerosis and decreases tissue cholesterol levels in a murine model of familial hypercholesterolemia
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The size of endothelial fenestrae in human liver sinusoids: implications for hepatocyte-directed gene transfer
E. Wisse et al.
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Species differences in transgene DNA uptake in hepatocytes after adenoviral transfer correlate with the size of endothelial fenestrae
J. Snoeys et al.
GENE THERAPY (2007)
The size of sinusoidal fenestrae is a critical determinant of hepatocyte transduction after adenoviral gene transfer
J Lievens et al.
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Long-term stable expression of human apolipoprotein A-I mediated by helper-dependent adenovirus gene transfer inhibits atherosclerosis progression and remodels atherosclerotic plaques in a mouse model of familial hypercholesterolemia
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Induction of immune tolerance to coagulation factor IX antigen by in vivo hepatic gene transfer
F Mingozzi et al.
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Humoral immune response in mice against a circulating antigen induced by adenoviral transfer is strictly dependent on expression in antigen-presenting cells
BR De Geest et al.
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Persistent hepatic expression of human apo A-I after transfer with a helper-virus independent adenoviral vector
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GENE THERAPY (2002)
Transgenic overexpression of human lecithin: cholesterol acyltransferase (LCAT) in mice does not increase aortic cholesterol deposition
JW Furbee et al.
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Effect of promoters and enhancers on expression, transgene DNA persistence, and hepatotoxicity after adenoviral gene transfer of human apolipoprotein A-I
S Van Linthout et al.
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Sustained expression of human apo A-I following adenoviral gene transfer in mice
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GENE THERAPY (2001)
Effects of cholesterol diets on vascular function and atherogenesis in rabbits
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Overexpression of human lecithin:cholesterol acyltransferase in mice offers no protection against diet-induced atherosclerosis
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APMIS (2000)
LCAT modulates atherogenic plasma lipoproteins and the extent of atherosclerosis only in the presence of normal LDL receptors in transgenic rabbits
ME Brousseau et al.
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