期刊
FREE RADICAL RESEARCH
卷 46, 期 4, 页码 460-478出版社
TAYLOR & FRANCIS LTD
DOI: 10.3109/10715762.2012.655730
关键词
OGG1; NEIL1,2,3; NTH1; glycosylase-deficient mice; metabolic disease; MUTYH
资金
- NCI NIH HHS [P01 CA160032] Funding Source: Medline
- NIDDK NIH HHS [R01 DK075974] Funding Source: Medline
This review will present a current understanding of mechanisms for the initiation of base excision repair (BER) of oxidatively-induced DNA damage and the biological consequences of deficiencies in these enzymes in mouse model systems and human populations.
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