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The shuttling SR protein 9G8 plays a role in translation of unspliced mRNA containing a constitutive transport element
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SR protein 9G8 modulates splicing of tau exon 10 via its proximal downstream intron, a clustering region for frontotemporal dementia mutations
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Tau-mediated neurodegeneration in Alzheimer's disease and related disorders
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Cellular tau pathology and immunohistochemical study of tau isoforms in sporadic tauopathies
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Tau exon 10, whose missplicing causes frontotemporal dementia, is regulated by an intricate interplay of cis elements and trans factors
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A minimal length between tau exon 10 and 11 is required for correct splicing of exon 10
QM Yu et al.
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Regulation of alternative splicing of human tau exon 10 by phosphorylation of splicing factors
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Hyperphosphorylation induces self-assembly of τ into tangles of paired helical filaments/straight filaments
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Splicing factors SRp20 and 9G8 promote the nucleocytoplasmic export of mRNA
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Complex regulation of tau exon 10, whose missplicing causes frontotemporal dementia
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