4.6 Review

Programmed necrosis and necroptosis signalling

相关参考文献

注意:仅列出部分参考文献,下载原文获取全部文献信息。
Review Cell Biology

IAPs, regulators of innate immunity and inflammation

Yann Estornes et al.

SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY (2015)

Article Biochemistry & Molecular Biology

Structural Study of the RIPoptosome Core Reveals a Helical Assembly for Kinase Recruitment

Tae-ho Jang et al.

BIOCHEMISTRY (2014)

Article Biochemistry & Molecular Biology

RIPK1 Regulates RIPK3-MLKL-Driven Systemic Inflammation and Emergency Hematopoiesis

James A. Rickard et al.

Article Biochemistry & Molecular Biology

RIPK1 Blocks Early Postnatal Lethality Mediated by Caspase-8 and RIPK3

Christopher P. Dillon et al.

Article Biochemistry & Molecular Biology

RIPK1-and RIPK3-induced cell death mode is determined by target availability

W. D. Cook et al.

CELL DEATH AND DIFFERENTIATION (2014)

Editorial Material Cell Biology

MLKL regulates necrotic plasma membrane permeabilization

Lorenzo Galluzzi et al.

CELL RESEARCH (2014)

Article Biochemistry & Molecular Biology

Necrosis-dependent and independent signaling of the RIP kinases in inflammation

Kenta Moriwaki et al.

CYTOKINE & GROWTH FACTOR REVIEWS (2014)

Article Biochemistry & Molecular Biology

RIPK1 can function as an inhibitor rather than an initiator of RIPK3-dependent necroptosis

Conor J. Kearney et al.

FEBS JOURNAL (2014)

Article Multidisciplinary Sciences

RIPK1 ensures intestinal homeostasis by protecting the epithelium against apoptosis

Nozomi Takahashi et al.

NATURE (2014)

Article Multidisciplinary Sciences

RIPK1 maintains epithelial homeostasis by inhibiting apoptosis and necroptosis

Marius Dannappel et al.

NATURE (2014)

Review Cell Biology

Regulated necrosis: the expanding network of non-apoptotic cell death pathways

Tom Vanden Berghe et al.

NATURE REVIEWS MOLECULAR CELL BIOLOGY (2014)

Review Medicine, General & Internal

Necroptosis

Andreas Linkermann et al.

NEW ENGLAND JOURNAL OF MEDICINE (2014)

Article Multidisciplinary Sciences

RIP1 suppresses innate immune necrotic as well as apoptotic cell death during mammalian parturition

William J. Kaiser et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2014)

Review Cell Biology

Necroptosis, in vivo detection in experimental disease models

Sandrine Jouan-Lanhouet et al.

SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY (2014)

Review Cell Biology

Molecular mechanisms of regulated necrosis

Lorenzo Galluzzi et al.

SEMINARS IN CELL & DEVELOPMENTAL BIOLOGY (2014)

Article Biochemistry & Molecular Biology

Positive and negative phosphorylation regulates RIP1-and RIP3-induced programmed necrosis

Thomas McQuade et al.

BIOCHEMICAL JOURNAL (2013)

Article Biochemistry & Molecular Biology

Intrinsic cleavage of receptor-interacting protein kinase-1 by caspase-6

B. J. van Raam et al.

CELL DEATH AND DIFFERENTIATION (2013)

Review Cardiac & Cardiovascular Systems

Targeting cell death in the reperfused heart: Pharmacological approaches for cardioprotection

Martinus I. F. J. Oerlemans et al.

INTERNATIONAL JOURNAL OF CARDIOLOGY (2013)

Article Biochemistry & Molecular Biology

Toll-like Receptor 3-mediated Necrosis via TRIF, RIP3, and MLKL

William J. Kaiser et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2013)

Article Biochemistry & Molecular Biology

Fas/CD95-Induced Chemokines Can Serve as Find-Me Signals for Apoptotic Cells

Sean P. Cullen et al.

MOLECULAR CELL (2013)

Article Multidisciplinary Sciences

Two independent pathways of regulated necrosis mediate ischemia-reperfusion injury

Andreas Linkermann et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2013)

Article Pharmacology & Pharmacy

Cobalt triggers necrotic cell death and atrophy in skeletal C2C12 myotubes

Francesca Rovetta et al.

TOXICOLOGY AND APPLIED PHARMACOLOGY (2013)

Article Cell Biology

TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1

D. M. Moujalled et al.

CELL DEATH & DISEASE (2013)

Article Cell Biology

Protective Roles for Caspase-8 and cFLIP in Adult Homeostasis

Ricardo Weinlich et al.

CELL REPORTS (2013)

Article Cell Biology

cFLIP Regulates Skin Homeostasis and Protects against TNF-Induced Keratinocyte Apoptosis

Diana Panayotova-Dimitrova et al.

CELL REPORTS (2013)

Article Biochemistry & Molecular Biology

Ferroptosis: An Iron-Dependent Form of Nonapoptotic Cell Death

Scott J. Dixon et al.

Review Biochemistry & Molecular Biology

Molecular definitions of cell death subroutines: recommendations of the Nomenclature Committee on Cell Death 2012

L. Galluzzi et al.

CELL DEATH AND DIFFERENTIATION (2012)

Article Biochemistry & Molecular Biology

cIAP1 and cIAP2 limit macrophage necroptosis by inhibiting Rip1 and Rip3 activation

S. McComb et al.

CELL DEATH AND DIFFERENTIATION (2012)

Article Biochemistry & Molecular Biology

TRAIL induces necroptosis involving RIPK1/RIPK3-dependent PARP-1 activation

S. Jouan-Lanhouet et al.

CELL DEATH AND DIFFERENTIATION (2012)

Article Biochemistry & Molecular Biology

Inhibition of Autophagy Rescues Palmitic Acid-induced Necroptosis of Endothelial Cells

Muhammad Jadoon Khan et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2012)

Article Biochemistry & Molecular Biology

Necrostatin-1 Suppresses Autophagy and Apoptosis in Mice Traumatic Brain Injury Model

Yao-Qi Wang et al.

NEUROCHEMICAL RESEARCH (2012)

Article Multidisciplinary Sciences

Staurosporine Induces Necroptotic Cell Death under Caspase-Compromised Conditions in U937 Cells

Zsuzsanna A. Dunai et al.

PLOS ONE (2012)

Article Cell Biology

Survival Function of the FADD-CASPASE-8-cFLIPL Complex

Christopher P. Dillon et al.

CELL REPORTS (2012)

Review Neurosciences

Programmed Necrosis: A Prominent Mechanism of Cell Death following Neonatal Brain Injury

Raul Chavez-Valdez et al.

NEUROLOGY RESEARCH INTERNATIONAL (2012)

Article Immunology

Mechanisms of necroptosis in T cells

Irene L. Ch'en et al.

JOURNAL OF EXPERIMENTAL MEDICINE (2011)

Article Biochemistry & Molecular Biology

The Ripoptosome, a Signaling Platform that Assembles in Response to Genotoxic Stress and Loss of IAPs

Tencho Tenev et al.

MOLECULAR CELL (2011)

Article Multidisciplinary Sciences

Caspase-8 regulates TNF-α-induced epithelial necroptosis and terminal ileitis

Claudia Guenther et al.

NATURE (2011)

Article Multidisciplinary Sciences

RIP3 mediates the embryonic lethality of caspase-8-deficient mice

William J. Kaiser et al.

NATURE (2011)

Article Multidisciplinary Sciences

Catalytic activity of the caspase-8-FLIPL complex inhibits RIPK3-dependent necrosis

Andrew Oberst et al.

NATURE (2011)

Article Microbiology

Virus Inhibition of RIP3-Dependent Necrosis

Jason W. Upton et al.

CELL HOST & MICROBE (2010)

Review Cell Biology

Molecular mechanisms of necroptosis: an ordered cellular explosion

Peter Vandenabeele et al.

NATURE REVIEWS MOLECULAR CELL BIOLOGY (2010)

Article Microbiology

Shigella Induces Mitochondrial Dysfunction and Cell Death in Nonmyleoid Cells

Leticia A. M. Carneiro et al.

CELL HOST & MICROBE (2009)

Article Cell Biology

Cellular IAPs inhibit a cryptic CD95-induced cell death by limiting RIP1 kinase recruitment

Peter Geserick et al.

JOURNAL OF CELL BIOLOGY (2009)

Article Immunology

Caspase-8 deficiency in epidermal keratinocytes triggers an inflammatory skin disease

Andrew Kovalenko et al.

JOURNAL OF EXPERIMENTAL MEDICINE (2009)

Review Biochemistry & Molecular Biology

A Tangled Web of Ubiquitin Chains: Breaking News in TNF-R1 Signaling

Katiuscia Bianchi et al.

MOLECULAR CELL (2009)

Article Biochemistry & Molecular Biology

IAP antagonists target cIAP1 to induce TNFα- dependent apoptosis

James E. Vince et al.

Article Biochemistry & Molecular Biology

Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury

A Degterev et al.

NATURE CHEMICAL BIOLOGY (2005)

Review Immunology

The caspase-8 modulator c-FLIP

T Kataoka

CRITICAL REVIEWS IN IMMUNOLOGY (2005)

Article Biochemistry & Molecular Biology

Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

O Micheau et al.