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The Sonic hedgehog pathway independently controls the patterning, proliferation and survival of neuroepithelial cells by regulating Gli activity
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Loss of the retrograde motor for IFT disrupts localization of Smo to cilia and prevents the expression of both activator and repressor functions of Gli
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Gli2 and Gli3 localize to cilia and require the intra-flagellar transport protein polaris for processing and function
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Mouse intraflagellar transport proteins regulate both the activator and repressor functions of Gli transcription factors
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Loss of cell polarity causes severe brain dysplasia in Lgl1 knockout mice
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Sonic hedgehog is required for progenitor cell maintenance in telencephalic stem cell niches
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