期刊
DEVELOPMENTAL CELL
卷 15, 期 4, 页码 494-496出版社
CELL PRESS
DOI: 10.1016/j.devcel.2008.09.016
关键词
-
资金
- Worldwide Cancer Research [03-0900] Funding Source: Medline
New work by Lehembre et al. in The EMBO Journal reveals that the cell-adhesion molecule, NCAM, is at the heart of crosstalk between E-cadherin loss and reciprocal focal adhesion assembly during the epithelial to mesenchymal transition (EMT). NCAM upregulation induces the formation of novel signaling complexes that correlate with NCAM-dependent focal adhesion assembly, migration, and cancer cell invasion.
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