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Mode of action: Developmental thyroid hormone insufficiency - Neurological abnormalities resulting from exposure to propylthiouracil
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Mode of action: Angiotensin-converting enzyme inhibition - Developmental effects associated with exposure to ACE inhibitors
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Mode of action: Disruption of brain cell replication, second messenger, and neurotransmitter systems during development leading to cognitive dysfunction - Developmental neurotoxicity of nicotine
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Overview: Using mode of action and life stage information to evaluate the human relevance of animal toxicity data
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Mode of action: Reduction of testosterone availability - Molinate-induced inhibition of spermatogenesis
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Mode of action: Inhibition of androgen receptor function - Vinclozolin-induced malformations in reproductive development
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Mode of action: Yolk sac poisoning and impeded histiotrophic nutrition - HBOC-related congenital malformations
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Mode of action: Impaired fetal Leydig cell function - Effects on male reproductive development produced by certain phthalate esters
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Mode of action: Neurotoxicity induced by thyroid hormone disruption during development - Hearing loss resulting from exposure to PHAHs
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Mode of action: Oxalate crystal-induced renal tubule degeneration and glycolic acid-induced dysmorphogenesis - Renal and developmental effects of ethylene glycol
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