期刊
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY
卷 89, 期 -, 页码 160-167出版社
ELSEVIER SCI LTD
DOI: 10.1016/j.yjmcc.2015.10.014
关键词
CaMKII; ox-CaMKII; MsrA; Heart failure; Myocardial infarction; Atrial fibrillation; Sinus node dysfunction; Atherosclerosis; Vascular smooth muscle; Asthma; Cancer
资金
- International Society for Heart Research
- National Institutes of Health (NIH) [R01-HL079031, R01-HL096652, R01-HL070250, R01-HL071140]
CaMKII is activated by oxidation of methionine residues residing in the regulatory domain. Oxidized CaMKII (ox-CaMKII) is now thought to participate in cardiovascular and pulmonary diseases and cancer. This invited review summarizes current evidence for the role of ox-CaMKII in disease, considers critical knowledge gaps and suggests new areas for inquiry. (C) 2015 Elsevier Ltd. All rights reserved.
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据
分享论文
