期刊
TRENDS IN CARDIOVASCULAR MEDICINE
卷 12, 期 1, 页码 46-49出版社
ELSEVIER SCIENCE LONDON
DOI: 10.1016/S1050-1738(01)00138-4
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资金
- NATIONAL INSTITUTE OF GENERAL MEDICAL SCIENCES [R01GM056904] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE ON AGING [R01AG014563] Funding Source: NIH RePORTER
- NIA NIH HHS [AG14563] Funding Source: Medline
- NIGMS NIH HHS [GM56904] Funding Source: Medline
beta-Adrenergic receptors transduce signals through the heterotrimeric G protein Galphas to regulate cardiac function. Recently, it has been discovered that Galphas could directly stimulate the Src family tyrosine kinase activity. This novel signaling pathway provides insights into the cAMP-PKA-independent signaling mechanisms used by beta-adrenergic receptors to regulate apoptosis, receptor desensitization, and other physiological functions. (C) 2002, Elsevier Science Inc.
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