期刊
ONCOLOGY
卷 62, 期 -, 页码 38-42出版社
KARGER
DOI: 10.1159/000048274
关键词
hepatocellular carcinoma; hepatitis B virus; hepatitis C virus; hypercarcinogenic state; liver cirrhosis; inflammation
类别
In spite of great efforts in the research relating to human hepatocarcinogenesis, its mechanism on the molecular level is yet to be determined. Chronic viral hepatitis may increase the chances of genetic events in hepatocytes of the host, by increasing the number of target cells or through proliferation of initiated hepatocytes, toward the eventual development of hepatocellular carcinoma. These conditions are referred to comprehensively as the hypercarcinogenic state. Our goal, then, should be directed to the reversion of the hypercarcinogenic state to the normoor hypocarcinogenic state so as to hopefully prevent or at least postpone the development of hepatocellular carcinoma. Copyright (C) 2002 S. Karger AG, Basel.
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据