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注意:仅列出部分参考文献,下载原文获取全部文献信息。Human RECQ1 Is a DNA Damage Responsive Protein Required for Genotoxic Stress Resistance and Suppression of Sister Chromatid Exchanges
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RECQL5/Recql5 helicase regulates homologous recombination and suppresses tumor formation via disruption of Rad51 presynaptic filaments
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RecQ helicases queuing with Srs2 to disrupt Rad51 filaments and suppress recombination
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Molecular genetics of RecQ helicase disorders
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Mechanisms of RecQ helicases in pathways of DNA metabolism and maintenance of genomic stability
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JOURNAL OF BIOLOGICAL CHEMISTRY (2006)
BLAP75/RMI1 promotes the BLM-dependent dissolution of homologous recombination intermediates
L Wu et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)
Initiation of DNA replication requires the RECQL4 protein mutated in Rothmund-Thomson syndrome
MN Sangrithi et al.
CELL (2005)
Recq15 and blm RecQ DNA helicases have nonredundant roles in suppressing crossovers
YD Hu et al.
MOLECULAR AND CELLULAR BIOLOGY (2005)
Examination of the roles of Sgs1 and Srs2 helicases in the enforcement of recombination fidelity in Saccharomyces cerevisiae
RM Spell et al.
GENETICS (2004)
DNA polymerase stabilization at stalled replication forks requires Mec1 and the RecQ helicase Sgs1
JA Cobb et al.
EMBO JOURNAL (2003)
The Srs2 helicase prevents recombination by disrupting Rad51 nucleoprotein filaments
X Veaute et al.
NATURE (2003)