期刊
CELL HOST & MICROBE
卷 3, 期 6, 页码 344-347出版社
CELL PRESS
DOI: 10.1016/j.chom.2008.05.010
关键词
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资金
- Howard Hughes Medical Institute Funding Source: Medline
According to a classical view of bacterial-host interactions at intestinal surfaces, the commensal microbiota establishes tolerance, and invasive pathogens cause stereotypic inflammation. The reality is more complex, marked by a menage A trois situation encompassing three emerging concepts: (1) pathogens take advantage of inflammation to cross the epithelial barrier, (2) pathogens reduce the commensal flora to invade their niche, and (3) pathogens express dedicated effectors that modulate inflammation.
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