4.7 Article

RIPK1-and RIPK3-induced cell death mode is determined by target availability

相关参考文献

注意:仅列出部分参考文献,下载原文获取全部文献信息。
Article Biochemistry & Molecular Biology

A robust methodology to subclassify pseudokinases based on their nucleotide-binding properties

James M. Murphy et al.

BIOCHEMICAL JOURNAL (2014)

Article Biochemistry & Molecular Biology

Positive and negative phosphorylation regulates RIP1-and RIP3-induced programmed necrosis

Thomas McQuade et al.

BIOCHEMICAL JOURNAL (2013)

Review Cell Biology

RIP3: a molecular switch for necrosis and inflammation

Kenta Moriwaki et al.

GENES & DEVELOPMENT (2013)

Article Cell Biology

TNF can activate RIPK3 and cause programmed necrosis in the absence of RIPK1

D. M. Moujalled et al.

CELL DEATH & DISEASE (2013)

Review Biochemistry & Molecular Biology

Many stimuli pull the necrotic trigger, an overview

N. Vanlangenakker et al.

CELL DEATH AND DIFFERENTIATION (2012)

Editorial Material Biochemistry & Molecular Biology

The flick of a switch: which death program to choose?

P. Vandenabeele et al.

CELL DEATH AND DIFFERENTIATION (2012)

Review Cell Biology

Signal transduction by tumor necrosis factor receptors

Lucia Cabal-Hierro et al.

CELLULAR SIGNALLING (2012)

Article Multidisciplinary Sciences

Mixed lineage kinase domain-like is a key receptor interacting protein 3 downstream component of TNF-induced necrosis

Jie Zhao et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2012)

Article Biochemistry & Molecular Biology

The Ripoptosome, a Signaling Platform that Assembles in Response to Genotoxic Stress and Loss of IAPs

Tencho Tenev et al.

MOLECULAR CELL (2011)

Article Multidisciplinary Sciences

RIP3 mediates the embryonic lethality of caspase-8-deficient mice

William J. Kaiser et al.

NATURE (2011)

Article Cell Biology

Caspase 8 inhibits programmed necrosis by processing CYLD

Marie Anne O'Donnell et al.

NATURE CELL BIOLOGY (2011)

Article Biochemistry & Molecular Biology

Inducible Dimerization and Inducible Cleavage Reveal a Requirement for Both Processes in Caspase-8 Activation

Andrew Oberst et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2010)

Review Cell Biology

Molecular mechanisms of necroptosis: an ordered cellular explosion

Peter Vandenabeele et al.

NATURE REVIEWS MOLECULAR CELL BIOLOGY (2010)

Article Biochemistry & Molecular Biology

Receptor Interacting Protein Kinase-3 Determines Cellular Necrotic Response to TNF-α

Sudan He et al.

Article Biochemistry & Molecular Biology

TNF-α induces two distinct caspase-8 activation pathways

Lai Wang et al.

Article Biochemistry & Molecular Biology

Cytoplasmic p53 is not required for PUMA-induced apoptosis

B. A. Callus et al.

CELL DEATH AND DIFFERENTIATION (2008)

Article Biochemistry & Molecular Biology

IAP antagonists target cIAP1 to induce TNFα- dependent apoptosis

James E. Vince et al.

Article Multidisciplinary Sciences

Surface comparison of active and inactive protein kinases identifies a conserved activation mechanism

Alexandr P. Kornev et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)

Article Biochemistry & Molecular Biology

Chemical inhibitor of nonapoptotic cell death with therapeutic potential for ischemic brain injury

A Degterev et al.

NATURE CHEMICAL BIOLOGY (2005)

Article Biochemistry & Molecular Biology

Q-VD-OPh, a broad spectrum caspase inhibitor with potent antiapoptotic properties

TM Caserta et al.

APOPTOSIS (2003)

Article Biochemistry & Molecular Biology

Induction of TNF receptor I-mediated apoptosis via two sequential signaling complexes

O Micheau et al.