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Casey M. Clements et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)
Mutational analysis of DJ-1 in Drosophila implicates functional inactivation by oxidative damage and aging
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Proper SUMO-1 conjugation is essential to DJ-1 to exert its full activities
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Inactivation of Drosophila DJ-1 leads to impairments of oxidative stress response and phosphatidylinositol 3-kinase/Akt signaling
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Interaction of DJ-1 with Daxx inhibits apoptosis signal-regulating kinase 1 activity and cell death
E Junn et al.
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DJ-1, a novel regulator of the tumor suppressor PTEN
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Familial Parkinson's disease-associated L166P mutation disrupts DJ-1 protein folding and function
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JOURNAL OF BIOLOGICAL CHEMISTRY (2004)
Differential effects of Parkinson's disease-associated mutations on stability and folding of DJ-1
K Görner et al.
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DJ-1 (PARK7) mutations are less frequent than Parkin (PARK2) mutations in early-onset Parkinson disease
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DJ-1 has a role in antioxidative stress to prevent cell death
T Taira et al.
EMBO REPORTS (2004)
The Parkinson's disease protein DJ-1 is neuroprotective due to cysteine-sulfinic acid-driven mitochondrial localization
RM Canet-Avilés et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2004)
Down regulation of DJ-1 enhances cell death by oxidative stress, ER stress, and proteasome inhibition
T Yokota et al.
BIOCHEMICAL AND BIOPHYSICAL RESEARCH COMMUNICATIONS (2003)
L166P mutant DJ-1, causative for recessive Parkinson's disease, is degraded through the ubiquitin-proteasome system
DW Miller et al.
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The role of pathogenic DJ-1 mutations in Parkinson's disease
PM Abou-Sleiman et al.
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Crystal structure of human DJ-1, a protein associated with early onset Parkinson's disease
X Tao et al.
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The crystal structure of DJ-1, a protein related to male fertility and bParkinson's disease
K Honbou et al.
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The 1.1-Å resolution crystal structure of DJ-1, the protein mutated in autosomal recessive early onset Parkinson's disease
MA Wilson et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2003)
Early-onset Parkinson's disease caused by a compound heterozygous DJ-1 mutation
S Hague et al.
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Mutations in the DJ-1 gene associated with autosomal recessive early-onset parkinsonism
V Bonifati et al.
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The DJ-1(L166P) mutant protein associated with early onset Parkinson's disease is unstable and forms higher-order protein complexes
MG Macedo et al.
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A missense mutation (L166P) in DJ-1, linked to familial Parkinson's disease, confers reduced protein stability and impairs homo-oligomerization
DJ Moore et al.
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Exchange of N-CoR corepressor and Tip60 coactivator complexes links gene expression by NF-κB and β-amyloid precursor protein
SH Baek et al.
CELL (2002)
Gene transfer of the JNK interacting protein-1 protects dopaminergic neurons in the MPTP model of Parkinson's disease
XG Xia et al.
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Dopaminergic cell death induced by MPP+, oxidant and specific neurotoxicants shares the common molecular mechanism
HS Chun et al.
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Hsp72 functions as a natural inhibitory protein of c-Jun N-terminal kinase
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Oxidized forms of peroxiredoxins and DJ-1 on two-dimensional gels increased in response to sublethal levels of paraquat
A Mitsumoto et al.
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DJ-1 is an indicator for endogenous reactive oxygen species elicited by endotoxin
A Mitsumoto et al.
FREE RADICAL RESEARCH (2001)
Nitric oxide negatively regulates c-Jun N-terminal kinase/stress-activated protein kinase by means of S-nitrosylation
HS Park et al.
PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2000)
Interaction among mitochondria, mitogen-activated protein kinases, and nuclear factor-κB in cellular models of Parkinson's disease
DS Cassarino et al.
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MPTP activates c-Jun NH2-terminal kinase (JNK) and its upstream regulatory kinase MKK4 in nigrostriatal neurons in vivo
MS Saporito et al.
JOURNAL OF NEUROCHEMISTRY (2000)