期刊
IMMUNITY
卷 20, 期 1, 页码 5-16出版社
CELL PRESS
DOI: 10.1016/S1074-7613(03)00356-X
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资金
- NIAID NIH HHS [AI49320, AI46629, R01 AI017672, AI17672, AI46578, P01 AI046629, R01 AI046578, P01 AI049320, R37 AI017672] Funding Source: Medline
- NIAMS NIH HHS [R01 AR035506, AR35506] Funding Source: Medline
- NIDDK NIH HHS [DK32520, P30 DK032520] Funding Source: Medline
- NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES [R01AI017672, R01AI046578, R37AI017672, P01AI049320, P01AI046629] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF ARTHRITIS AND MUSCULOSKELETAL AND SKIN DISEASES [R01AR035506] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES [P30DK032520] Funding Source: NIH RePORTER
Virus-specific memory T cell populations demonstrate plasticity in antigenic and functional phenotype, in recognition of antigen, and in their ability to accommodate new memory T cell populations. The adaptability of complex antigen-specific T cell repertoires allows the host to respond to a diverse array of pathogens and accommodate memory pools to many pathogens in a finite immune system. This is in part accounted for by crossreactive memory T cells, which can be employed in immune responses and mediate protective immunity or life-threatening immunopathology.
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