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M. A. Hassan Talukder et al.
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Xiang-Dong Li et al.
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Alessandro Bellis et al.
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S. Frantz et al.
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Adrian T. Gonon et al.
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Phosphorylation of endothelial nitric-oxide synthase regulates superoxide generation from the enzyme
Chun-An Chen et al.
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Melatonin receptor-mediated protection against myocardial ischaemia/reperfusion injury: role of its anti-adrenergic actions
Sonia Genade et al.
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Signaling pathways in ischemic preconditioning
James M. Downey et al.
HEART FAILURE REVIEWS (2007)
Myocardial ischemia results in tetrahydrobiopterin (BH4) oxidation with impaired endothelial function ameliorated by BH4
Cristian Dumitrescu et al.
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Regulation of endothelial and myocardial NO synthesis by multi-site eNOS phosphorylation
Peter F. Mount et al.
JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2007)
Cardiomyocyte-specific overexpression of NO synthase-3 protects against myocardial ischemia-reperfusion injury
John W. Elrod et al.
ARTERIOSCLEROSIS THROMBOSIS AND VASCULAR BIOLOGY (2006)
PI 3-kinase, protein kinase C, and protein kinase A are involved in the trigger phase of β1-adrenergic preconditioning
A Robinet et al.
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New directions for protecting the heart against ischaemia-reperfusion injury: targeting the Reperfusion Injury Salvage Kinase (RISK)-pathway
DJ Hausenloy et al.
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Early preconditioning prevents the loss of endothelial nitric oxide synthase and enhances its activity in the ischemic/reperfused rat heart
C Muscari et al.
LIFE SCIENCES (2004)
Ischemic preconditioning attenuates calpain-mediated degradation of structural proteins through a protein kinase A-dependent mechanism
J Inserte et al.
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Protein kinase A as another mediator of ischemic preconditioning independent of protein kinase C
S Sanada et al.
CIRCULATION (2004)
Nitric-oxide synthase (NOS) reductase domain models suggest a new control element in endothelial NOS that attenuates calmodulin-dependent activity
GM Knudsen et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2003)
Site-specific dephosphorylation of endothelial nitric oxide synthase by protein phosphatase 2A: Evidence for crosstalk between phosphorylation sites
DM Greif et al.
BIOCHEMISTRY (2002)
Shear stress stimulates phosphorylation of endothelial nitric-oxide synthase at Ser1179 by Akt-independent mechanisms -: Role of protein kinase A
YC Boo et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2002)
The contribution of endothelial nitric oxide synthase to early ischaemic preconditioning: the lowering of the preconditioning threshold. An investigation in eNOS knockout mice
RM Bell et al.
CARDIOVASCULAR RESEARCH (2001)
Coordinated control of endothelial nitric-oxide synthase phosphorylation by protein kinase C and the cAMP-dependent protein kinase
BJ Michell et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2001)
Acute modulation of endothelial Akt/PKB activity alters nitric oxide-dependent vasomotor activity in vivo
ZY Luo et al.
JOURNAL OF CLINICAL INVESTIGATION (2000)
Exogenous nitric oxide can trigger a preconditioned state through a free radical mechanism, but endogenous nitric oxide is not a trigger of classical ischemic preconditioning
A Nakano et al.
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Enhanced electron flux and reduced calmodulin dissociation may explain calcium-independent eNOS activation by phosphorylation
TJ McCabe et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)