4.7 Article

Ablation of junctin or triadin is associated with increased cardiac injury following ischaemia/reperfusion

相关参考文献

注意:仅列出部分参考文献,下载原文获取全部文献信息。
Article Biochemistry & Molecular Biology

ER stress modulates cellular metabolism

Xiaoli Wang et al.

BIOCHEMICAL JOURNAL (2011)

Article Biochemistry & Molecular Biology

Structure of the Ire1 autophosphorylation complex and implications for the unfolded protein response

Maruf M. U. Ali et al.

EMBO JOURNAL (2011)

Review Biochemistry & Molecular Biology

Tearin' Up My Heart: Proteolysis in the Cardiac Sarcomere

Andrea L. Portbury et al.

JOURNAL OF BIOLOGICAL CHEMISTRY (2011)

Review Physiology

Cell Death in the Pathogenesis of Heart Disease: Mechanisms and Significance

Russell S. Whelan et al.

ANNUAL REVIEW OF PHYSIOLOGY (2010)

Article Biochemistry & Molecular Biology

Calpains and proteasomes mediate degradation of ryanodine receptors in a model of cardiac ischemic reperfusion

Zully Pedrozo et al.

BIOCHIMICA ET BIOPHYSICA ACTA-MOLECULAR BASIS OF DISEASE (2010)

Article Cardiac & Cardiovascular Systems

Transient Ca2+ depletion of the sarcoplasmic reticulum at the onset of reperfusion

Carlos A. Valverde et al.

CARDIOVASCULAR RESEARCH (2010)

Review Cardiac & Cardiovascular Systems

The SR-mitochondria interaction: a new player in cardiac pathophysiology

Marisol Ruiz-Meana et al.

CARDIOVASCULAR RESEARCH (2010)

Article Cardiac & Cardiovascular Systems

Polyol pathway impairs the function of SERCA and RyR in ischemic-reperfused rat hearts by increasing oxidative modifications of these proteins

Wai Ho Tang et al.

JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2010)

Article Cardiac & Cardiovascular Systems

Calpain translocation and activation as pharmacological targets during myocardial ischemia/reperfusion

Victor Hernando et al.

JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2010)

Article Biochemistry & Molecular Biology

Molecular Symbiosis of CHOP and C/EBPβ Isoform LIP Contributes to Endoplasmic Reticulum Stress-Induced Apoptosis

Calin-Bogdan Chiribau et al.

MOLECULAR AND CELLULAR BIOLOGY (2010)

Article Multidisciplinary Sciences

Ablation of triadin causes loss of cardiac Ca2+ release units, impaired excitation-contraction coupling, and cardiac arrhythmias

Nagesh Chopra et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2009)

Article Dentistry, Oral Surgery & Medicine

XBP1 May Determine the Size of the Ameloblast Endoplasmic Reticulum

M. Tsuchiya et al.

JOURNAL OF DENTAL RESEARCH (2008)

Article Cardiac & Cardiovascular Systems

Expression of SERCA isoform with faster Ca2+ transport properties improves postischemic cardiac function and Ca2+ handling and decreases myocardial infarction

M. A. Hassan Talukder et al.

AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)

Article Cardiac & Cardiovascular Systems

Overexpression of histidine-rich Ca-binding protein protects against ischemia/reperfusion-induced cardiac injury

Xiaoyang Zhou et al.

CARDIOVASCULAR RESEARCH (2007)

Article Cardiac & Cardiovascular Systems

On the role of junctin in cardiac Ca2+ handling, contractility, and heart failure

Ulrich Gergs et al.

AMERICAN JOURNAL OF PHYSIOLOGY-HEART AND CIRCULATORY PHYSIOLOGY (2007)

Review Biochemistry & Molecular Biology

Connecting endoplasmic reticulum stress to autophagy by unfolded protein response and calcium

M. Hoyer-Hansen et al.

CELL DEATH AND DIFFERENTIATION (2007)

Review Medicine, Research & Experimental

Endoplasmic reticulum stress: cell life and death decisions

CY Xu et al.

JOURNAL OF CLINICAL INVESTIGATION (2005)

Article Cardiac & Cardiovascular Systems

Activation of connexin-43 hemichannels can elevate [Ca2+]i and [Na+]i in rabbit ventricular myocytes during metabolic inhibition

FG Li et al.

JOURNAL OF MOLECULAR AND CELLULAR CARDIOLOGY (2001)