期刊
INFECTION AND IMMUNITY
卷 73, 期 1, 页码 657-660出版社
AMER SOC MICROBIOLOGY
DOI: 10.1128/IAI.73.1.657-660.2005
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资金
- NATIONAL CANCER INSTITUTE [P30CA042014] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES [R01AI043521, R01AI024158, R29AI043521, R01AI032223] Funding Source: NIH RePORTER
- NCI NIH HHS [P30 CA042014, 5P30CA42014] Funding Source: Medline
- NIAID NIH HHS [R56 AI032223, R29 AI043521, R01 AI032223, AI24158, AI-32223, AI-43521, R01 AI024158, R01 AI043521] Funding Source: Medline
TLR2(-/-)/scid double-mutant mice were infected with B. burgdorferi to assess the relative importance of acquired and innate host defenses. Although spirochete levels at 4 weeks were lower in TLR2(-/-) mice than in TLR2(-/-)/scid mice, the increased arthritis severity of TLR2 (Toll-like receptor 2)-deficient mice was reduced by the presence of the scid mutation.
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