期刊
NEUROCHEMICAL RESEARCH
卷 32, 期 4-5, 页码 833-835出版社
SPRINGER/PLENUM PUBLISHERS
DOI: 10.1007/s11064-006-9221-9
关键词
genetics; amyloid; Alzheimer's disease
资金
- Intramural NIH HHS Funding Source: Medline
- Medical Research Council [G0701075] Funding Source: Medline
- MRC [G0701075] Funding Source: UKRI
In this review, I discuss the possibility that A beta 42 has a physiologic function in blood vessel homeostasis and the consequences that this might have for theories concerning the pathogenesis of Alzheimer's disease and for treatment.
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