期刊
CANCER PREVENTION RESEARCH
卷 3, 期 11, 页码 1379-1381出版社
AMER ASSOC CANCER RESEARCH
DOI: 10.1158/1940-6207.CAPR-10-0251
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资金
- NCI NIH HHS [R01 CA111536, R01 CA120388, CA11536, CA077646, R01 CA077646, CA120388] Funding Source: Medline
This perspective on Liby et al. (beginning on page 1427 in this issue of the journal) discusses the importance of the finding that two synthetic triterpenoids prolonged survival in a pancreatic cancer mouse model. This finding is significant because pancreatic cancer is one of the deadliest human cancers. These compounds inhibited the interaction between NF-kappa B and signal transducer and activator of transcription 3, and determining the mechanisms underlying this inhibition will help to rapidly move these compounds into phase I clinical trials. Cancer Prev Res; 3(11); 1379-81. (C) 2010 AACR.
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