期刊
TRENDS IN MICROBIOLOGY
卷 15, 期 3, 页码 119-126出版社
ELSEVIER SCIENCE LONDON
DOI: 10.1016/j.tim.2007.01.003
关键词
-
资金
- NATIONAL CANCER INSTITUTE [R01CA097093] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES [R01AI067952] Funding Source: NIH RePORTER
- NCI NIH HHS [CA097093] Funding Source: Medline
- NIAID NIH HHS [AI051686, AI067952] Funding Source: Medline
- Wellcome Trust [GR066559] Funding Source: Medline
During infection, viruses attempt to hijack the cell while the host responds with various defense systems. Traditional defenses include the interferon response and apoptosis, but recent work suggests that this antiviral arsenal also includes the cellular DNA damage response machinery. The observation of interactions between viruses and cellular DNA repair proteins has not only uncovered new complexities of the virus-host interaction but is also reinforcing the view that viruses can reveal key regulators of cellular pathways through the proteins they target.
作者
我是这篇论文的作者
点击您的名字以认领此论文并将其添加到您的个人资料中。
推荐
暂无数据