期刊
IMMUNOLOGIC RESEARCH
卷 37, 期 1, 页码 61-77出版社
HUMANA PRESS INC
DOI: 10.1007/BF02686090
关键词
Salmonella; enteritis; innate immunity; mucosa
类别
资金
- NATIONAL INSTITUTE OF ALLERGY AND INFECTIOUS DISEASES [R21AI065619, R01AI048815] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF DIABETES AND DIGESTIVE AND KIDNEY DISEASES [P30DK040561] Funding Source: NIH RePORTER
- NIAID NIH HHS [R01 AI048815, R01 AI 48815, R21 AI 965619, R21 AI065619] Funding Source: Medline
- NIDDK NIH HHS [P30 DK040561, P30 DK040561-11] Funding Source: Medline
Acute gastroenteritis caused by Salmonella typhimurium infection is a clinical problem with significant public health impact. The availability of several experimental models of this condition has allowed detailed investigation of the cellular and molecular interactions involved in its pathogenesis. Such studies have shed light on the roles played by bacterial virulence factors and host innate immune mechanisms in the development of intestinal inflammation.
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