期刊
CANCER CELL
卷 22, 期 1, 页码 5-6出版社
CELL PRESS
DOI: 10.1016/j.ccr.2012.06.006
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资金
- NHLBI NIH HHS [R37 HL052725, P01 HL048546] Funding Source: Medline
- NIDDK NIH HHS [R01 DK043889] Funding Source: Medline
DNA damage can arrest replication forks during S phase. Failure to stabilize and restart arrested forks results in fork collapse and genomic instability. In this issue of Cancer Cell, Schlacher et al. show that the Fanconi anemia and BRCA2 tumor suppressor pathways cooperate to protect stalled replication forks from degradation.
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