期刊
EMOTION
卷 8, 期 5, 页码 701-713出版社
AMER PSYCHOLOGICAL ASSOC
DOI: 10.1037/a0013447
关键词
anger; STAXI; psychopathology; EEG; brain asymmetry
资金
- NIDA NIH HHS [R21 DA14111, R21 DA014111] Funding Source: Medline
- NIMH NIH HHS [T32 MH019554, R01 MH061358, R01 MH061358-01, R01 MH61358, T32 MH19554] Funding Source: Medline
- NATIONAL INSTITUTE OF MENTAL HEALTH [R01MH061358, T32MH019554] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE ON DRUG ABUSE [R21DA014111] Funding Source: NIH RePORTER
Depression and anxiety often involve high levels of trait anger and disturbances in anger expression. Reported anger experience and outward anger expression have recently been associated with left-biased asymmetry of frontal cortical activity, assumed to reflect approach motivation. However, different styles of anger expression could presumably involve different brain mechanisms and/or interact with psychopathology to produce various patterns of brain asymmetry. The present study explored these issues by comparing resting regional electroencephalographic activity in participants high in trait anger who differed in anger expression style (high anger-in, high anger-out, both) and participants low in trait anger, with depression and anxiety systematically assessed. Trait anger, not anger-in or anger-out, predicted left-biased asymmetry at medial frontal EEG sites. The anger-in group reported higher levels of anxious apprehension than did the anger-out group, Furthermore, anxious apprehension moderated the relationship between trait anger, anger-in, and asymmetry in favor of the left hemisphere. Results suggest that motivational direction is not always the driving force behind the relationship of anger and left frontal asymmetry. Findings also support a distinction between anxious apprehension and anxious arousal.
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