期刊
CHILD DEVELOPMENT
卷 81, 期 1, 页码 340-356出版社
WILEY
DOI: 10.1111/j.1467-8624.2009.01398.x
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资金
- NICHD NIH HHS [R01 HD042608, R01 HD042608-07, R56 HD042608] Funding Source: Medline
- NIDA NIH HHS [P30 DA023920, P30 DA023920-01A1, P20 DA017592, P20 DA017592-050001] Funding Source: Medline
- NIMH NIH HHS [R01 MH054257, R01 MH054257-08] Funding Source: Medline
- EUNICE KENNEDY SHRIVER NATIONAL INSTITUTE OF CHILD HEALTH & HUMAN DEVELOPMENT [R56HD042608] Funding Source: NIH RePORTER
- EUNICE KENNEDY SHRIVER NATIONAL INSTITUTE OF CHILD HEALTH &HUMAN DEVELOPMENT [R01HD042608] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF MENTAL HEALTH [R01MH054257] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE ON DRUG ABUSE [P30DA023920, P20DA017592] Funding Source: NIH RePORTER
To further the understanding of the effects of early experiences, 9-month-old infants were observed during a frustration task. The analytical sample was composed of 348 linked triads of participants (adoptive parents, adopted child, and birth parent[s]) from a prospective adoption study. It was hypothesized that genetic risk for externalizing problems and affect dysregulation in the adoptive parents would independently and interactively predict a known precursor to externalizing problems: heightened infant attention to frustrating events. Results supported the moderation hypotheses involving adoptive mother affect dysregulation: Infants at genetic risk showed heightened attention to frustrating events only when the adoptive mother had higher levels of anxious and depressive symptoms. The Genotype x Environment interaction pattern held when substance use during pregnancy was considered.
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