相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Elevated plasma concentration of NO and cGMP may be responsible for the decreased platelet aggregation and platelet leukocyte conjugation in platelets hypo-responsive to catecholamines
Yean Kyoung Koo et al.
PLATELETS (2009)
Reduced GPIIb/IIIa expression in platelets hyposensitive to catecholamines when activated with TRAP
Jeong Mi Kim et al.
THROMBOSIS RESEARCH (2009)
The role of Akt in the signaling pathway of the, glycoprotein Ib-IX-induced platelet activation
Hong Yin et al.
BLOOD (2008)
Rottlerin: an inappropriate and ineffective inhibitor of PKC delta
Stephen P. Soltoff
TRENDS IN PHARMACOLOGICAL SCIENCES (2007)
Differential requirements for calcium and Src family kinases in platelet GPIIb/IIIa activation and thromboxane generation downstream of different G-protein pathways
RT Dorsam et al.
BLOOD (2005)
Impaired platelet responses to thrombin and collagen in AKT-1-deficient mice
JH Chen et al.
BLOOD (2004)
The protein kinase C inhibitor RO318220 potentiates thrombin-stimulated platelet-supported prothrombinase activity
FS London
BLOOD (2003)
Apparent heterogeneous responsiveness of human platelet rich plasma to catecholamines
MK Pyo et al.
PLATELETS (2003)
Roles for both cyclic GMP and cyclic AMP in the inhibition of collagen-induced platelet aggregation by nitroprusside
EK Jang et al.
BRITISH JOURNAL OF HAEMATOLOGY (2002)
Platelets in nonresponders to epinephrine stimulation showed reduced response to ADP
TK Nakahashi et al.
THROMBOSIS RESEARCH (2001)
The P2Y12 receptor induces platelet aggregation through weak activation of the αIIbβ3 integrin -: a phosphoinositide 3-kinase-dependent mechanism
G Kauffenstein et al.
FEBS LETTERS (2001)
α2A-adrenergic receptor stimulation potentiates calcium release in platelets by modulating cAMP levels
IMLW Keularts et al.
JOURNAL OF BIOLOGICAL CHEMISTRY (2000)