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Id2 intrinsically regulates lymphoid and erythroid development via interaction with different target proteins
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Gain-of-function mutation of GATA-2 in acute myeloid transformation of chronic myeloid leukemia
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Loss of β-catenin impairs the renewal of normal and CML stem cells in vivo
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Leukemia stem cells in a genetically defined murine model of blast-crisis CML
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SET-CAN, the product of the t(9;9) in acute undifferentiated leukemia, causes expansion of early hematopoietic progenitors and hyperproliferation of stomach mucosa in transgenic mice
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HOXA10 is a critical regulator for hematopoietic stem cells and erythroid/megakaryocyte development
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The tumor suppressor menin regulates hematopoiesis and myeloid transformation by influencing Hox gene expression
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PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2006)
Insertional mutagenesis identifies genes that promote the immortalization of primary bone marrow progenitor cells
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The tumor suppressor PP2A is functionally inactivated in blast crisis CML through the inhibitory activity of the BCR/ABL-regulated SET protein
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Murine hematopoietic stem cells change their surface phenotype during ex vivo expansion
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Tumor suppressor NM23-H1 is a granzyme A-activated DNase during CTL-mediated apoptosis, and the nucleosome assembly protein SET is its inhibitor
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Nup98-HoxA9 immortalizes myeloid progenitors, enforces expression of Hoxa9, Hoxa7 and Meis1, and alters cytokine-specific responses in a manner similar to that induced by retroviral co-expression of Hoxa9 and Meis1
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ONCOGENE (2001)
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Defining roles for HOX and MEIS1 genes in induction of acute myeloid leukemia
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