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J. Wang et al.
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CIP2A Is Associated with Human Breast Cancer Aggressivity
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Chronic myeloid leukemia patients with the e13a2 BCR-ABL fusion transcript have inferior responses to imatinib compared to patients with the e14a2 transcript
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Jak2 inhibition deactivates Lyn kinase through the SET-PP2A-SHP1 pathway, causing apoptosis in drug-resistant cells from chronic myelogenous leukemia patients
A. K. Samanta et al.
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CIP2A is overexpressed in gastric cancer and its depletion leads to impaired clonogenicity, senescence, or differentiation of tumor cells
Wenjuan Li et al.
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J. Ma et al.
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D. Perrotti et al.
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The tumor suppressor PP2A is functionally inactivated in blast crisis CML through the inhibitory activity of the BCR/ABL-regulated SET protein
P Neviani et al.
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D White et al.
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VJSA Lobo et al.
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L Wang et al.
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Analysis of relative gene expression data using real-time quantitative PCR and the 2-ΔΔCT method
KJ Livak et al.
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V Janssens et al.
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