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Newborn humans manifest autoantibodies to defined self molecules detected by antigen microarray informatics
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Over 20% of patients with chronic lymphocytic leukemia carry stereotyped receptors: pathogenetic implications and clinical correlations
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Unmutated and mutated chronic lymphocytic leukemias derive from self-reactive B cell precursors despite expressing different antibody reactivity
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Natural antibodies and the autoimmunity of atherosclerosis
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In vivo measurements document the dynamic cellular kinetics of chronic lymphocytic leukemia B cells
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Mechanisms of disease: Chronic lymphocytic leukemia
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A syntaxin 1, Gαo, and N-type calcium channel complex at a presynaptic nerve terminal:: Analysis by quantitative immunocolocalization
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Multiple distinct sets of stereotyped antigen receptors indicate a role for antigen in promoting chronic lymphocytic leukemia
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Subsets with restricted immunoglobulin gene rearrangement features indicate a role for antigen selection in the development of chronic lymphocytic leukemia
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Chronic lymphocytic leukemias utilizing the VH3-21 gene display highly restricted Vλ2-14 gene use and homologous CDR3s:: implicating recognition of a common antigen epitope
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ZAP-70 expression identifies a chronic lymphocytic leukemia subtype with unmutated immunoglobulin genes, inferior clinical outcome, and distinct gene expression profile
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Differential signaling via surface IgM is associated with VH gene mutational status and CD38 expression in chronic lymphocytic leukemia
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Evidence for involvement of a hydrophobic patch in framework region 1 of human v4-34-encoded igs in recognition of the red blood cell I antigen
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Relationship between p53 dysfunction, CD38 expression, and IgVH mutation in chronic lymphocytic leukemia
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BLOOD (2002)