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注意:仅列出部分参考文献,下载原文获取全部文献信息。Human Th17 cells: Are they different from murine Th17 cells?
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Natural agonists for aryl hydrocarbon receptor in culture medium are essential for optimal differentiation of Th17 T cells
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In vivo activated monocytes from the site of inflammation in humans specifically promote Th17 responses
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The strength of T cell stimulation determines IL-7 responsiveness, secondary expansion, and lineage commitment of primed human CD4(+)IL-7R(hi) T cells
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IL-23 Promotes Maintenance but Not Commitment to the Th17 Lineage
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TGF-β-induced Foxp3 inhibits TH17 cell differentiation by antagonizing RORγt function
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IL-21 and TGF-β are required for differentiation of human TH17 cells
Li Yang et al.
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Differentiation of human TH-17 cells does require TGF-β!
Anne O'Garra et al.
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Interactions among the transcription factors Runx1, RORγt and Foxp3 regulate the differentiation of interleukin 17-producing T cells
Fuping Zhang et al.
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A critical function for transforming growth factor-beta, interleukin 23 and proinflammatory cytokines in driving and modulating human T-H-17 responses
Elisabetta Volpe et al.
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The differentiation of human TH-17 cells requires transforming growth factor-β and induction of the nuclear receptor RORγt
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Optimal induction of T helper 17 cells in humans requires T cell receptor ligation in the context of Toll-like receptor-activated monocytes
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IL-21 initiates an alternative pathway to induce proinflammatory TH17 cells
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Activation-induced FOXP3 in human T effector cells does not suppress proliferation or cytokine production
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Transient expression of FOXP3 in human activated nonregulatory CD4(+) T cells
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Molecular mechanisms underlying FOXP3 induction in human T cells
Pierre-Yves Mantel et al.
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Interleukin 17-producing CD4+ effector T cells develop via a lineage distinct from the T helper type 1 and 2 lineages
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Treatment with a neutralizing anti-murine interleukin-17 antibody after the onset of collagen-induced arthritis reduces joint inflammation, cartilage destruction, and bone erosion
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Crucial role for nuclear factor of activated T cells in T cell receptor-mediated regulation of human interleukin-17
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Conversion of peripheral CD4+CD25- naive T cells to CD4+CD25+ regulatory T cells by TGF-β induction of transcription factor Foxp3
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Dendritic-cell control of pathogen-driven T-cell polarization
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T cell fitness determined by signal strength
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Is selection for TCR affinity a factor in cytokine polarization?
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The potency of TCR signaling differentially regulates NFATc/p activity and early IL-4 transcription in naive CD4+ T cells
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Microbial lipopeptides induce the production of IL-17 in Th cells
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