期刊
EUROPEAN JOURNAL OF PHARMACOLOGY
卷 405, 期 1-3, 页码 161-166出版社
ELSEVIER SCIENCE BV
DOI: 10.1016/S0014-2999(00)00549-5
关键词
stressful stimuli; neuropeptide; c-fos; paraventricular nucleus; supraoptic nucleus; amygdaloid nucleus; central
This short review summarizes the effect of various stressful stimuli on the expression of neuropeptides which co-localize in corticotrophin releasing hormone (CRH)-synthesizing neurons in the hypothalamic paraventricular nucleus, as well as in oxytocin and vasopressin neurons in the supraoptic nucleus. Stress-induced changes failed to act on CRH neurons in the central amygdaloid nucleus but formalin-evoked pain enhanced galanin mRNA expression in the medial subdivision of this nucleus. Changes in the expression of enkephalin. galanin, dynorphin and cholecystokinin mRNA in response to restraint and formalin-induced pain are documented in hypothalamic and amygdaloid nuclei by in situ hybridization histochemical technique. (C) 2000 Elsevier Science B.V. All rights reserved.
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