相关参考文献
注意:仅列出部分参考文献,下载原文获取全部文献信息。Structural diversity and endotoxic activity of the lipopolysaccharide of Yersinia pestis
S. V. Dentovskaya et al.
BIOCHEMISTRY-MOSCOW (2008)
Differential activation of human TLR4 by Escherichia coli and Shigella flexneri 2a lipopolysaccharide:: Combined effects of lipid A acylation state and TLR4 polymorphisms on signaling
Prasad Rallabhandi et al.
JOURNAL OF IMMUNOLOGY (2008)
Soluble CD14 discriminates slight structural differences between lipid as that lead to distinct host cell activation
Yasuyuki Asai et al.
JOURNAL OF IMMUNOLOGY (2007)
Effect of chemotype on Escherichia coli interactions with bactericidal proteins
S. V. Zubova et al.
BULLETIN OF EXPERIMENTAL BIOLOGY AND MEDICINE (2007)
Role of N-acetylglucosamine within core lipopolysaccharide of several species of gram-negative bacteria in targeting the DC-SIGN (CD209)
Pei Zhang et al.
JOURNAL OF IMMUNOLOGY (2006)
DC-SIGN induction in alveolar macrophages defines privileged target host cells for mycobacteria in patients with tuberculosis
L Tailleux et al.
PLOS MEDICINE (2005)
Influence of CD14 on ligand interactions between lipopolysaccharide and its receptor complex
SC Gangloff et al.
JOURNAL OF IMMUNOLOGY (2005)
Phospholipids inhibit lipopolysaccharide (LPS)-induced cell activation: A role for LPS-binding protein
M Mueller et al.
JOURNAL OF IMMUNOLOGY (2005)
Bartonella quintana lipopolysaccharide effects on leukocytes, CXC chemokines and apoptosis:: a study on the human whole blood and a rat model
G Matera et al.
INTERNATIONAL IMMUNOPHARMACOLOGY (2003)
Structural basis for endotoxic and antagonistic activities: investigation with novel synthetic lipid A analogs
S Kusumoto et al.
JOURNAL OF ENDOTOXIN RESEARCH (2003)
Endotoxic properties of lipid A from Comamonas testosteroni
K Tanamoto et al.
MICROBIOLOGY-SGM (2001)
Toll-like receptor 4, but not toll-like receptor 2, is a signaling receptor for Escherichia and Salmonella lipopolysaccharides
RI Tapping et al.
JOURNAL OF IMMUNOLOGY (2000)
Salmonella-type heptaacylated lipid A is inactive and acts as an antagonist of lipopolysaccharide action on human line cells
K Tanamoto et al.
JOURNAL OF IMMUNOLOGY (2000)