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Expression, biological activities and mechanisms of action of A20 (TNFAIP3)
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Enterocyte-specific A20 deficiency sensitizes to tumor necrosis factor-induced toxicity and experimental colitis
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African-Derived Genetic Polymorphisms in TNFAIP3 Mediate Risk for Autoimmunity
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The NF-κB negative regulator TNFAIP3 (A20) is inactivated by somatic mutations and genomic deletions in marginal zone lymphomas
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Coeliac disease-associated risk variants in TNFAIP3 and REL implicate altered NF-κB signalling
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Common and different genetic background for rheumatoid arthritis and coeliac disease
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Association of TNFAIP3 polymorphism with rheumatic heart disease in Chinese Han population
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TNFAIP3 (A20) is a tumor suppressor gene in Hodgkin lymphoma and primary mediastinal B cell lymphoma
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Mutations of multiple genes cause deregulation of NF-κB in diffuse large B-cell lymphoma
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Frequent inactivation of A20 in B-cell lymphomas
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A large-scale replication study identifies TNIP1, PRDM1, JAZF1, UHRF1BP1 and IL10 as risk loci for systemic lupus erythematosus
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Genome-wide association study in a Chinese Han population identifies nine new susceptibility loci for systemic lupus erythematosus
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Homeostatic MyD88-dependent signals cause lethal inflammation in the absence of A20
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JOURNAL OF EXPERIMENTAL MEDICINE (2008)
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Genetic variants near TNFAIP3 on 6q23 are associated with systemic lupus erythematosus
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Multiple polymorphisms in the TNFAIP3 region are independently associated with systemic lupus erythematosus
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Two independent alleles at 6q23 associated with risk of rheumatoid arthritis
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Genome-wide association study of 14,000 cases of seven common diseases and 3,000 shared controls
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