期刊
CURRENT OPINION IN IMMUNOLOGY
卷 13, 期 5, 页码 543-548出版社
CURRENT BIOLOGY LTD
DOI: 10.1016/S0952-7915(00)00256-9
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Several genetic defects in class-switch recombination, leading to hyper-IgM syndromes, have been recently described in humans. Besides the well-known role of interaction between CD40-ligand and CD40, these pathological conditions definitively demonstrate the requirement of CD40-mediated NF-kappaB activation and the essential role of a newly described molecule, activation-induced cytidine deaminase (AID), in B cell terminal differentiation.
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