期刊
CURRENT OPINION IN CELL BIOLOGY
卷 13, 期 5, 页码 578-583出版社
CURRENT BIOLOGY LTD
DOI: 10.1016/S0955-0674(00)00254-4
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- NIDDK NIH HHS [DK 54605] Funding Source: Medline
- NIGMS NIH HHS [GM 50194] Funding Source: Medline
Two groups have now reported the viability of mice that lack syndecan-4. These mice have wound healing/angiogenesis problems, and fibroblasts from these animals differ in adhesion and migration from normal. This is consistent with recent in vitro data indicating a need for signaling via syndecan-4 for focal adhesion formation, and reports that overexpression of proteins that bind syndecan-4 can modify cell adhesion and migration.
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