期刊
JOURNAL OF PEDIATRICS
卷 140, 期 2, 页码 267-269出版社
MOSBY-ELSEVIER
DOI: 10.1067/mpd.2002.121699
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A boy had infantile-onset systemic inflammation, growth failure, hepatosplenomegaly, anemia, leukocytopenia, progressive muscular dystrophy, and hypercalprotectinemia, resulting in marked hyperzincemia. His mother had a history of chronic arthritis since childhood and also showed hypercalprotectinemia/hyperzincemia. We postulate an inherent defect in calprotectin metabolism.
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