期刊
JOURNALS OF GERONTOLOGY SERIES A-BIOLOGICAL SCIENCES AND MEDICAL SCIENCES
卷 57, 期 7, 页码 B257-B269出版社
OXFORD UNIV PRESS INC
DOI: 10.1093/gerona/57.7.B257
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资金
- NIA NIH HHS [R01 AG07444] Funding Source: Medline
Forty years after its discovery, replicative senescence remains a rich source of information about cell-cycle regulation and the progression from a normal to a transformed phenotype. Effectors of this growth-arrested state are being discovered at a great pace. This review discusses the latest findings on the players responsible for establishing replicative senescence, as well as the associated telomere shortening.
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