期刊
JOURNAL OF DENTAL RESEARCH
卷 82, 期 7, 页码 509-513出版社
SAGE PUBLICATIONS INC
DOI: 10.1177/154405910308200704
关键词
alcohol drinking; epidemiology; periodontal diseases; periodontitis
资金
- NCI NIH HHS [CA55075] Funding Source: Medline
- NHLBI NIH HHS [HL35464] Funding Source: Medline
- NIAAA NIH HHS [AA11181] Funding Source: Medline
- NIDCR NIH HHS [DE12102] Funding Source: Medline
Alcohol consumption impairs neutrophil, macrophage, and T-cell functions, increasing the likelihood of infections. We examined the association between alcohol consumption and periodontitis, prospectively, among 39,461 male health professionals aged 40 to 75 years and free of periodontitis at the start of follow-up. Alcohol intake was assessed at baseline and updated every 4 years by a food-frequency questionnaire. Periodontal disease status was self-reported and validated against radiographs. Multivariate analysis was adjusted for age, smoking, diabetes, body-mass index, physical activity, time period, and caloric intake. During 406,160 person-years of follow-up, there were 2125 cases of periodontitis. Compared with non-drinkers, the relative risk (95% confidence interval) among men reporting usual alcohol intake of 0.1-4.9 g/day was 1.24 (1.09, 1.42); 5.0 to 14.9 g/day, 1.18 (1.04, 1.35); 15 to 29.9 g/day, 1.18 (1.01, 1.38); and greater than or equal to 30 g/day, 1.27 (1.08, 1.49). The results suggest that alcohol consumption is an independent modifiable risk factor for periodontitis.
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