期刊
NATURE REVIEWS MOLECULAR CELL BIOLOGY
卷 4, 期 8, 页码 651-657出版社
NATURE PUBLISHING GROUP
DOI: 10.1038/nrm1173
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Many agonists of G-protein-coupled receptors (GPCRs) can stimulate receptor tyrosine kinases and the extracellular signal-regulated kinase (ERK)/mitogen-activated protein kinase (MAPK) pathway. A 'transactivation' mechanism, which links these events in one signalling chain, inspired many researchers, but inevitably raised new questions. A 'multi-track' model for GPCR signalling to the ERK/MAPK pathway might resolve some of the puzzles in the transactivation field.
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