期刊
JOURNAL OF VIROLOGY
卷 78, 期 14, 页码 7852-7860出版社
AMER SOC MICROBIOLOGY
DOI: 10.1128/JVI.78.14.7852-7860.2004
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资金
- NCI NIH HHS [K01 CA095443] Funding Source: Medline
- NIAID NIH HHS [R37 AI14910, R37 AI014910] Funding Source: Medline
Superantigens are microbial proteins that strongly stimulate T cells. We described previously that the Epstein-Barr virus (EBV) transactivates a superantigen encoded by the human endogenous retrovirus, HERV-K18. We now report that the transactivation is dependent upon the EBV latent cycle proteins. Moreover, LMP-2A is sufficient for induction of HERV-K18 superantigen activity.
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