期刊
PEST MANAGEMENT SCIENCE
卷 61, 期 4, 页码 397-401出版社
JOHN WILEY & SONS LTD
DOI: 10.1002/ps.996
关键词
pyrethroids; resistance; esterases; Helicoverpa armigera; piperonyl butoxide
Pyrethroid resistance in field populations of Australian Helicoverpa armigera (Hubner) is primarily a consequence of the overproduction of esterase isoenzymes which metabolise and sequester pyrethroid insecticides. Biochemical studies have shown that pyrethroid-resistance-associated esterases in H armigera are inhibited by the insecticide synergist piperonyl butoxide (PBO). Esterase inhibition by PBO did not occur immediately after dosing, but exhibited maximum inhibition 3-4h after dosage. Esterase activity subsequently recovered until full activity was restored by 24 h. Topical bioassays using a pre-treatment of PBO showed that maximum H armigera mortality was achieved with pre-treatment times corresponding to maximum esterase inhibition. These results demonstrated that, with correct temporal application, PBO can restore pyrethroid efficacy against H armigera. It would also be expected that restoration of efficacy with other conventional insecticides, currently compromised by esterase-based resistance mechanisms, would occur. circle star 2004 Society of Chemical Industry.
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