期刊
NATURE NEUROSCIENCE
卷 8, 期 7, 页码 839-841出版社
NATURE PUBLISHING GROUP
DOI: 10.1038/nn0705-839
关键词
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Long-term potentiation and long-term depression require postsynaptic depolarization, which many current models attribute to backpropagating action potentials. New experimental work suggests, however, that other mechanisms can lead to dendritic depolarization, and that backpropagating action potentials may be neither necessary nor sufficient for synaptic plasticity in vivo.
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