期刊
NATURE NEUROSCIENCE
卷 8, 期 7, 页码 862-863出版社
NATURE PUBLISHING GROUP
DOI: 10.1038/nn1474
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资金
- NICHD NIH HHS [HD29891] Funding Source: Medline
- NIMH NIH HHS [KO2-MH01188, P50-MH64445] Funding Source: Medline
We evaluated signal-noise discrimination in children with and without dyslexia, using magnocellular and parvocellular visual stimuli presented either with or without high noise. Dyslexic children had elevated contrast thresholds when stimuli of either type were presented in high noise, but performed as well as non-dyslexic children when either type was displayed without noise. Our findings suggest that deficits in noise exclusion, not magnocellular processing, contribute to the etiology of dyslexia.
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