期刊
INFECTION AND IMMUNITY
卷 73, 期 10, 页码 7006-7010出版社
AMER SOC MICROBIOLOGY
DOI: 10.1128/IAI.73.10.7006-7010.2005
关键词
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资金
- NIAID NIH HHS [U54 AI057160] Funding Source: Medline
Pathophysiological changes associated with anthrax lethal toxin included loss of plasma proteins, decreased platelet count, slower clotting times, fibrin deposits in tissue sections, and gross and histopathological evidence of hemorrhage. These findings suggest that blood vessel leakage and hemorrhage lead to disseminating intravascular coagulation and/or circulatory shock as an underlying pathophysiological mechanism.
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