期刊
ANNUAL REVIEW OF PHYSIOLOGY
卷 72, 期 -, 页码 81-94出版社
ANNUAL REVIEWS
DOI: 10.1146/annurev-physiol-021909-135931
关键词
Forkhead; Akt signaling; stress response; cardiac hypertrophy
类别
资金
- NHLBI NIH HHS [R01 HL065619, R37 HL065619, HL65619] Funding Source: Medline
- NIA NIH HHS [AG024282, P01 AG024282] Funding Source: Medline
- NIEHS NIH HHS [P30 ES010126] Funding Source: Medline
- NIGMS NIH HHS [R01 GM061728-07, GM61728, R01 GM061728, R01 GM061728-08, R01 GM061728-09A1] Funding Source: Medline
- NATIONAL HEART, LUNG, AND BLOOD INSTITUTE [R37HL065619, R01HL065619] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF ENVIRONMENTAL HEALTH SCIENCES [P30ES010126] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE OF GENERAL MEDICAL SCIENCES [R01GM061728] Funding Source: NIH RePORTER
- NATIONAL INSTITUTE ON AGING [P01AG024282] Funding Source: NIH RePORTER
The Forkhead family of transcription factors mediates many aspects of physiology, including stress response, metabolism, commitment to apoptosis, and development. The Forkhead box subfamily O (FoxO) proteins have garnered particular interest due to their involvement in the modulation of cardiovascular biology. In this review, we discuss the mechanisms of Fox regulation and outcomes of FoxO signaling under normal and pathological cardiovascular contexts.
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