期刊
DEVELOPMENT AND PSYCHOPATHOLOGY
卷 17, 期 1, 页码 145-165出版社
CAMBRIDGE UNIV PRESS
DOI: 10.1017/S095457940505008X
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资金
- NIAAA NIH HHS [AA09367, R01 AA009367, AA00175] Funding Source: Medline
- NIDA NIH HHS [DA05147, R01 DA013240, R37 DA005147, R01 DA005147, DA13240] Funding Source: Medline
- NIMH NIH HHS [R21 MH065137-05, R21 MH065137-04, R21 MH065137, T32 MH017069, R21 MH065137-01, MH 65137, R21 MH065137-03, MH17069, R21 MH065137-02] Funding Source: Medline
The present study attempted to determine the direction and etiology of the robust relationship between childhood externalizing (EXT) symptoms and parent-child conflict using a genetically informative longitudinal model and data from the ongoing Minnesota Twin Family Study. Participants consisted of 1,506 same-sex twins assessed at ages 11 and 14. and their parents. The relationship between EXT and parent-child conflict from ages 11 to 14 was examined within a biometrical cross-lagged design. The results revealed three primary findings: first, the stability of conflict and externalizing over time is largely, although not solely, a result of genetic factors. Second, there appears to he a bidirectional relationship between conflict and EXT over time, Such that both conflict and EXT at 11 independently predict the other 3 years later. Finally, the results are consistent with the notion that parent-child conflict partially results from parental responses to their child's heritable externalizing behavior. while simultaneously Contributing to child externalizing via environmental mechanisms. These results suggest a downward spiral of interplay between parent-child conflict and EXT, and offer confirmation of a (partially) environmentally mediated effect of parenting on child behavior.
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