4.7 Article

Expression of FcRL4 defines a pro-inflammatory, RANKL-producing B cell subset in rheumatoid arthritis

相关参考文献

注意:仅列出部分参考文献,下载原文获取全部文献信息。
Article Rheumatology

Rituximab abrogates joint destruction in rheumatoid arthritis by inhibiting osteoclastogenesis

Maria J. H. Boumans et al.

ANNALS OF THE RHEUMATIC DISEASES (2012)

Article Rheumatology

Cytokine mRNA profiling identifies B cells as a major source of RANKL in rheumatoid arthritis

Lorraine Yeo et al.

ANNALS OF THE RHEUMATIC DISEASES (2011)

Article Medicine, Research & Experimental

Attenuation of HIV-associated human B cell exhaustion by siRNA down regulation of inhibitory receptors

Lela Kardava et al.

JOURNAL OF CLINICAL INVESTIGATION (2011)

Article Immunology

Discriminating gene expression profiles of memory B cell subpopulations

Goetz R. A. Ehrhardt et al.

JOURNAL OF EXPERIMENTAL MEDICINE (2008)

Review Biochemistry & Molecular Biology

IL-6, RANKL, TNF-alpha/IL-1: interrelations in bone resorption pathophysiology

KT Steeve et al.

CYTOKINE & GROWTH FACTOR REVIEWS (2004)

Article Multidisciplinary Sciences

The inhibitory potential of Fc receptor homolog 4 on memory B cells

GRA Ehrhardt et al.

PROCEEDINGS OF THE NATIONAL ACADEMY OF SCIENCES OF THE UNITED STATES OF AMERICA (2003)

Article Immunology

Immunostimulatory DNA sequences influence the course of adjuvant arthritis

A Ronaghy et al.

JOURNAL OF IMMUNOLOGY (2002)