期刊
NEUROLOGY
卷 67, 期 8, 页码 1486-1489出版社
LIPPINCOTT WILLIAMS & WILKINS
DOI: 10.1212/01.wnl.0000240066.02404.e6
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资金
- NIDA NIH HHS [DA12065] Funding Source: Medline
- NIMH NIH HHS [MH45294, MH 62512, MH59745, MH58164] Funding Source: Medline
The cellular basis for cognitive deficits in HIV+ patients with and without a history of methamphetamine (METH) use is unclear. We found that HIV+ METH users had more severe loss of interneurons that was associated with cognitive impairment. Compared with other markers, loss of calbindin and parvalbumin interneurons in the frontal cortex was the most significant correlate to memory deficits, suggesting a role in neurobehavioral alterations of HIV+ METH users.
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