期刊
BIOCHEMICAL SOCIETY TRANSACTIONS
卷 35, 期 -, 页码 120-123出版社
PORTLAND PRESS LTD
DOI: 10.1042/BST0350120
关键词
genetic disease; knockout animal; spontaneous mutation; transgenic animal; transient receptor potential gene (TRP gene); transient receptor potential melastatin 8 (TRPM8)
The identification of the biological functions of TRP (transient receptor potential) proteins requires genetic approaches because a selective TRP channel pharmacology to unravel the roles of TRPs is not available so far for most TRPs. A survey is therefore presented of transgenic animal models carrying mutations in TRP genes, as well as of those TRP genes that when mutated result in human disease; the chromosomal locations of TRIP channel genes in the human and mouse are also presented.
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